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Hypoglycemia and the sympathoadrenal system: Neurogenic symptoms are largely the result of sympathetic neural, rather than adrenomedullary, activation
Michael A. DeRosa
, Philip E. Cryer
Institute of Clinical and Translational Sciences (ICTS)
Division of Endocrinology, Metabolism & Lipid Research
Research output
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Contribution to journal
›
Article
›
peer-review
109
Scopus citations
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Dive into the research topics of 'Hypoglycemia and the sympathoadrenal system: Neurogenic symptoms are largely the result of sympathetic neural, rather than adrenomedullary, activation'. Together they form a unique fingerprint.
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Keyphrases
Hypoglycemia
100%
Sympathoadrenal System
100%
Hyperinsulinemic-euglycemic Clamp
50%
Neural Activation
33%
Relative Contribution
16%
Control Subjects
16%
Diastolic Blood Pressure
16%
Healthy Controls
16%
Epinephrine
16%
Hyperinsulinemic
16%
Hyperglycemic Clamp
16%
Hemodynamic Response
16%
Adrenal Medulla
16%
Hemodynamic Changes
16%
Sympathetic Nervous System
16%
Symptom Score
16%
Heart Rate
16%
Two-component
16%
Euglycemic
16%
Norepinephrine Spillover
16%
Forearm Blood Flow
16%
Subject Control
16%
Neuroscience
Hypoglycemia
100%
Norepinephrine
66%
Blood Plasma
50%
Epinephrine
16%
Adrenal Medulla
16%
Sympathetic Nervous System
16%