Hypertension and prolonged vasoconstrictor signaling in RGS2-deficient mice

Keyphrases

• Hypertension 100%
• Gene-deficient Mice 100%
• Vasodilator 100%
• RGS2 100%
• Agonist Efficacy 33%
• Receptor-mediated 16%
• G Protein-coupled Receptor 16%
• Blood Pressure Regulation 16%
• Blood Pressure 16%
• GTPase-activating Protein 16%
• Ca2+ Signaling 16%
• Cardiac Output 16%
• Neurotransmitters 16%
• Constriction 16%
• Vasculature 16%
• Vascular Smooth muscle Cells 16%
• G Protein 16%
• Prolonged Response 16%
• Genetic Defects 16%
• Novel Risk Factors 16%
• P2Y Receptors 16%
• Renovascular 16%
• G Protein-coupled 16%
• Deactivation Rate 16%
• G Protein-coupled Receptor Signaling 16%
• Hypertensive Phenotypes 16%
• Resistance Vasculature 16%
• Kinetic Control 16%
• Signal Termination 16%

Neuroscience

• Vasoconstrictors 100%
• In Vitro 66%
• G Protein Coupled Receptor 66%
• In Vivo 33%
• Guanosine Triphosphatase Activating Protein 33%
• Calcium Signaling 33%
• Agonist 33%
• Vascular Smooth Muscle 33%
• G Protein 33%
• Neurotransmitter 33%
• Termination Signal 33%
• P2Y Receptor 33%
• Heterotrimeric G Protein 33%
• Receptor 33%
• Risk Factor 33%

Biochemistry, Genetics and Molecular Biology

• RGS2 100%
• G Protein Coupled Receptor 50%
• Vascularization 50%
• Genetics 25%
• Calcium Signaling 25%
• Blood Pressure 25%
• Agonist 25%
• GTPase-activating Protein 25%
• Neurotransmitter 25%
• Heterotrimeric G Protein 25%
• Heart Output 25%
• Blood Pressure Regulation 25%
• P2Y Receptor 25%
• G Protein 25%