Abstract

In humans, a considerable fraction of the retinoid pool in skin is derived from vitamin A2 (all-trans 3,4-dehydroretinal). Vitamin A2 may be locally generated by keratinocytes, which can convert vitamin A1 (all-trans retinol) into vitamin A2 in cell culture. We report that human cytochrome P450 (hP450) 27C1, a previously 'orphan' enzyme, can catalyze this reaction. Purified recombinant hP450 27C1 bound and desaturated all-trans retinol, retinal, and retinoic acid, as well as 11-cis-retinal. Although the physiological role of 3,4-dehydroretinoids in humans is unclear, we have identified hP450 27C1 as an enzyme capable of efficiently mediating their formation.

Original languageEnglish
Pages (from-to)1304-1312
Number of pages9
JournalFEBS Letters
Volume590
Issue number9
DOIs
StatePublished - May 1 2016

Keywords

  • Cytochrome P450
  • desaturation
  • mass spectrometry
  • retinoids
  • spectroscopy

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