Haemophilus influenzae from patients with chronic obstructive pulmonary disease exacerbation induce more inflammation than colonizers

Keyphrases

• Inflammation 100%
• Haemophilus Influenzae 100%
• Acute Exacerbation of COPD (AECOPD) 100%
• Colonizer 100%
• Airway 66%
• Airway Inflammation 50%
• Antibody Response 33%
• Chronic Obstructive Pulmonary Disease 33%
• Airway Infection 33%
• Non-associated 16%
• Mouse Model 16%
• Nuclear factor-B 16%
• Virulence 16%
• Interleukin-8 16%
• Epithelial Cells 16%
• Airway Epithelial Cells 16%
• P38 Mitogen-activated Protein Kinase (p38 MAPK) 16%
• Bacterial Infection 16%
• In Vivo Model 16%
• Serum Antibody 16%
• Neutrophil Recruitment 16%
• Clinical Symptoms 16%
• Gene Pathway 16%
• New Strain 16%
• Bacterial Strains 16%
• Airway Function 16%
• Symptom Worsening 16%
• In Vitro Cell Models 16%
• Primary Human Airway Epithelial Cells 16%
• Defense Signaling 16%
• Defense Genes 16%
• Signaling Pathway Activation 16%
• Bacterial Adherence 16%
• Influenza Strains 16%
• Bacterial Defense 16%
• Adherence Genes 16%

Pharmacology, Toxicology and Pharmaceutical Science

• Inflammation 100%
• Chronic Obstructive Lung Disease 100%
• Disease Exacerbation 100%
• Haemophilus influenzae 100%
• Respiratory Tract Inflammation 50%
• Mouse Model 33%
• Respiratory Tract Infection 33%
• Alternative Complement Pathway C3 C5 Convertase 16%
• Nuclear Factor 16%
• Cell Culture 16%
• Bacterial Infection 16%
• Interleukin 8 16%
• Mitogen Activated Protein Kinase P38 16%

Immunology and Microbiology

• Haemophilus influenzae 100%
• Airway 100%
• Infection 25%
• Mouse Model 16%
• Antibody Response 16%
• Complement Factor B 8%
• Mitogen 8%
• Cell Culture 8%
• Neutrophil 8%
• Antibody Blood Level 8%
• Bacterial Strain 8%
• Bacterium Adherence 8%
• Interleukin 8 8%
• In Vitro 8%