Growing pains: The cytoskeleton as a critical regulator of pain plasticity

Gautam Bhave; Robert W. Gereau IV

doi:10.1016/S0896-6273(03)00500-2

Growing pains: The cytoskeleton as a critical regulator of pain plasticity

Gautam Bhave, Robert W. Gereau IV

Research output: Contribution to journal › Review article › peer-review

27 Scopus citations

Abstract

Inflammatory mediators act on peripheral sensory neurons to produce pain and hypersensitivity after tissue injury. In this issue of Neuron, Dina et al. report that inflammatory mediators, such as epinephrine and prostaglandins, appear to couple to specific G protein-coupled receptor signaling pathways through plastic interactions with the cytoskeleton.

Original language	English
Pages (from-to)	577-579
Number of pages	3
Journal	Neuron
Volume	39
Issue number	4
DOIs	https://doi.org/10.1016/S0896-6273(03)00500-2
State	Published - Aug 14 2003

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title = "Growing pains: The cytoskeleton as a critical regulator of pain plasticity",

abstract = "Inflammatory mediators act on peripheral sensory neurons to produce pain and hypersensitivity after tissue injury. In this issue of Neuron, Dina et al. report that inflammatory mediators, such as epinephrine and prostaglandins, appear to couple to specific G protein-coupled receptor signaling pathways through plastic interactions with the cytoskeleton.",

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AB - Inflammatory mediators act on peripheral sensory neurons to produce pain and hypersensitivity after tissue injury. In this issue of Neuron, Dina et al. report that inflammatory mediators, such as epinephrine and prostaglandins, appear to couple to specific G protein-coupled receptor signaling pathways through plastic interactions with the cytoskeleton.

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Growing pains: The cytoskeleton as a critical regulator of pain plasticity

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