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GRK mythology: G-protein receptor kinases in cardiovascular disease
Gerald W. Dorn
Roy and Diana Vagelos Division of Biology & Biomedical Sciences (DBBS)
Institute of Clinical and Translational Sciences (ICTS)
Department of Medicine
Division of Pharmacogenomics
DBBS - Molecular Cell Biology
DBBS - Human and Statistical Genetics
DBBS - Molecular Genetics and Genomics
Hope Center for Neurological Disorders
Research output
:
Contribution to journal
›
Review article
›
peer-review
84
Scopus citations
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Keyphrases
Cardiovascular Disease
100%
G Protein-coupled Receptor Kinase
100%
Mythology
100%
Heart Failure
28%
Neurohormone
28%
Signaling Pathway
14%
G Protein-coupled Receptor
14%
Receptor Downregulation
14%
Myocardial Infarction
14%
Kinase Function
14%
Receptor Desensitization
14%
Receptor Phosphorylation
14%
Cardiac Hypertrophy
14%
Protective Behavior
14%
Differential Effects
14%
Neurohormonal
14%
GRK2
14%
Kinase Signaling
14%
Functional Pathways
14%
Adverse Consequences
14%
Medicine and Dentistry
Receptor
100%
Cardiovascular Disease
100%
Kinase
100%
Guanine Nucleotide Binding Protein
100%
Congestive Heart Failure
22%
Receptor down Regulation
22%
Neurohormone
22%
Myocardial Infarction
11%
G Protein Coupled Receptor
11%
Ventricular Hypertrophy
11%
Cell Signaling Pathway
11%
Pharmacology, Toxicology and Pharmaceutical Science
Receptor
100%
Guanine Nucleotide Binding Protein
100%
Cardiovascular Disease
100%
Congestive Heart Failure
20%
Neurohormone
20%
Heart Infarction
10%
Desensitization
10%
Heart Ventricle Hypertrophy
10%
G Protein Coupled Receptor
10%
Neuroscience
G Protein
100%
Receptor
100%
Receptor down Regulation
25%
Neurohormone
25%
G Protein Coupled Receptor
12%
Receptor Phosphorylation
12%
Myocardial Infarction
12%
Biochemistry, Genetics and Molecular Biology
G Protein
100%
Receptor down Regulation
28%
G Protein Coupled Receptor
14%