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Glutamate and GABA receptor dysfunction in the fetal alcohol syndrome
J. W. Olney
, D. F. Wozniak
, V. Jevtovic-Todorovic
,
N. B. Farber
, P. Bittigau
, C. Ikonomidou
Institute of Clinical and Translational Sciences (ICTS)
Department of Psychiatry
Division of Education
Research output
:
Contribution to journal
›
Article
›
peer-review
59
Scopus citations
Overview
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Keyphrases
Glutamate Receptors
100%
GABAA Receptor (GABAAR)
100%
Ethanol
100%
Fetal Alcohol Spectrum Disorder
100%
NMDA Antagonist
83%
Brain Development
33%
Apoptotic Neurodegeneration
33%
Brain Growth Spurt
33%
Rodents
16%
Ketamine
16%
Obstetrics
16%
Anticonvulsant
16%
Hyperactivity
16%
Human Development
16%
Dual Mechanism
16%
Angels
16%
Anesthesiology
16%
Nitrous Oxide
16%
Pregnant Women
16%
Brain Mass
16%
Neurobehavior
16%
Synaptogenesis
16%
Suicide
16%
Benzodiazepines
16%
Phencyclidine
16%
Barbiturates
16%
Human Fetus
16%
Paediatric Neurology
16%
General Anesthetics
16%
Damaging Effects
16%
Apoptogenic
16%
Dust
16%
CNS Neurons
16%
Human Environment
16%
NMDA Glutamate Receptors
16%
Action-based
16%
Laughing Gas
16%
Pharmacology, Toxicology and Pharmaceutical Science
Fetal Alcohol Syndrome
100%
N Methyl Dextro Aspartic Acid Receptor Blocking Agent
100%
Glutamate Receptor
100%
4 Aminobutyric Acid Receptor
100%
4 Aminobutyric Acid Receptor Stimulating Agent
100%
Nerve Degeneration
40%
Phencyclidine
40%
Nitrous Oxide
40%
Receptor
20%
Anticonvulsive Agent
20%
Barbiturate
20%
Ketamine
20%
N Methyl Dextro Aspartic Acid
20%
Neuroscience
Glutamate Receptor
100%
NMDA Receptor Antagonist
100%
Fetal Alcohol Spectrum Disorder
100%
GABA Receptor
100%
Neurodegeneration
40%
N-Methyl-D-Aspartic Acid
20%
GABAA Receptor
20%
Nitrous Oxide
20%
Synaptogenesis
20%
Barbiturate
20%
Ketamine
20%
Phencyclidine
20%
Pediatric Neurology
20%
Anticonvulsant
20%