Glucocorticoids differentially regulate degradation of MyoD and Id1 by N-terminal ubiquitination to promote muscle protein catabolism

Keyphrases

• Muscle Protein 100%
• MyoD 100%
• Protein Catabolism 100%
• N-terminal Ubiquitination 100%
• Ubiquitination Pathway 60%
• Protein Degradation 40%
• N-terminus 40%
• Muscle Regeneration 40%
• Muscle Development 40%
• Myotubes 40%
• MyoD Protein 40%
• Glucocorticoid Receptor 20%
• Cellular Compartments 20%
• Human Disease 20%
• Disease States 20%
• Negative Regulator 20%
• Lysine 20%
• Ubiquitination 20%
• Pharmacological Treatment 20%
• Ubiquitin-proteasome System 20%
• Id Proteins 20%
• Ubiquitin System 20%
• Regulatory Factors 20%
• Protein Abundance 20%
• Master Switches 20%
• Accelerated Degradation 20%
• Muscle Protein Degradation 20%
• Skeletal muscle Wasting 20%

Biochemistry, Genetics and Molecular Biology

• N-Terminus 100%
• Protein Catabolism 100%
• Ubiquitination 100%
• MyoD 100%
• Glucocorticoid 100%
• Ubiquitin 33%
• Myogenesis 33%
• Muscle Regeneration 33%
• MyoD Protein 33%
• Skeletal Muscle 16%
• Glucocorticoid Receptor 16%
• Proteasome 16%
• Inhibitor of Differentiation 1 16%
• Lysine 16%
• Drug Therapy 16%