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Genetic Discovery of ATP-Sensitive K
+
Channels in Cardiovascular Diseases
Yan Huang
, Dan Hu
, Congxin Huang
,
Colin G. Nichols
Department of Cell Biology & Physiology
Center for the Investigation of Membrane Excitability Diseases
Roy and Diana Vagelos Division of Biology & Biomedical Sciences (DBBS)
Institute of Clinical and Translational Sciences (ICTS)
DBBS - Molecular Cell Biology
DBBS - Neurosciences
DBBS - Biochemistry, Biophysics, and Structural Biology
Research output
:
Contribution to journal
›
Review article
›
peer-review
37
Scopus citations
Overview
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+
Channels in Cardiovascular Diseases'. Together they form a unique fingerprint.
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Keyphrases
Cardiovascular Disease
100%
KATP Channel
100%
K+ Channels
100%
Phenotypic Drug Discovery
100%
Pathophysiology
33%
Pancreatic Cells
33%
Genetic Changes
33%
Protein Complex
33%
SUR1
33%
Myocytes
33%
Genomic Analysis
33%
Physiological Processes
33%
Cardiovascular System
33%
Pathological Process
33%
Causal Relationship
33%
Hetero
33%
Membrane Excitability
33%
Cellular Metabolism
33%
Pore-forming
33%
Genetically Modified Organisms
33%
Biochemistry, Genetics and Molecular Biology
Genetics
100%
Adenosine Triphosphate
100%
Potassium Channel
100%
Gene Mutation
25%
Sulfonylurea Receptor
25%
Excitability
25%
Animal Model
25%
Protein Complexes
25%
Cell Metabolism
25%
Genetically Modified Animal
25%
Genomics
25%
Adenosine Triphosphate Sensitive Potassium Channel
25%
Pharmacology, Toxicology and Pharmaceutical Science
Adenosine Triphosphate
100%
Potassium Channel
100%
Cardiovascular Disease
100%
Pathophysiology
25%
Potassium Ion
25%
Sulfonylurea Receptor
25%
Adenosine Triphosphate Sensitive Potassium Channel
25%
Transgenic Animal
25%