Abstract

SNF2-family DNA translocases, a large family of ATPases, have poorly defined roles in genomic stability. In a recent study, Feng et al. identified a synthetic lethal interaction between the SNF2 translocase SMARCAL1 and Fanconi anemia (FA) group M (FANCM), revealing a new genetic buffering mechanism that maintains genome stability by aiding DNA replication at loci enriched in simple repeats.

Original languageEnglish
Pages (from-to)266-267
Number of pages2
JournalTrends in Genetics
Volume41
Issue number4
DOIs
StatePublished - Apr 2025

Keywords

  • SNF2-family DNA translocases
  • simple repeats

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