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Gene therapy for carcinoma of the breast genetic ablation strategies
D. T. Curiel
Roy and Diana Vagelos Division of Biology & Biomedical Sciences (DBBS)
Institute of Clinical and Translational Sciences (ICTS)
Siteman Cancer Center
Bursky Center for Human Immunology & Immunotherapy Programs (CHiiPs)
Department of Radiation Oncology
Center of Regenerative Medicine
DBBS - Developmental, Regenerative and Stem Cell Biology
DBBS - Immunology
DBBS - Molecular Microbiology and Microbial Pathogenesis
DBBS - Molecular Genetics and Genomics
Brain Tumor Center
Research output
:
Contribution to journal
›
Review article
›
peer-review
8
Scopus citations
Overview
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Keyphrases
Gene Therapy
100%
Genetic Ablation
100%
Oncogene
100%
Breast Carcinoma
100%
Ablation Strategy
100%
Dysregulated
50%
ErbB2
50%
Estrogen Receptor
50%
Transcriptional Level
50%
Expression Level
50%
Antisense
50%
Oncoprotein
50%
Neoplastic Transformation
50%
Single-chain Antibody
50%
Human Trials
50%
Cellular Control
50%
Functional Knockout
50%
Molecular Lesions
50%
Cyclin D1 (CCND1)
50%
Mutation Compensation
50%
Therapy Strategy
50%
Ribozyme
50%
Molecular Intervention
50%
Modal Systems
50%
Medicine and Dentistry
Carcinoma
100%
Gene Therapy
100%
Oncogene
100%
Estrogen Receptor
50%
Antisense
50%
Single Chain Fragment Variable Antibody
50%
Cyclin D1
50%
Oncoprotein
50%
Ribozyme
50%
Diseases
50%
Biochemistry, Genetics and Molecular Biology
Gene Therapy
100%
Genetic Ablation
100%
Oncogene
100%
Estrogen Receptor
50%
Antisense
50%
Single-Chain Variable Fragment
50%
Cyclin D1
50%
Ribozyme
50%
Molecular Lesion
50%
Molecular Intervention
50%
Pharmacology, Toxicology and Pharmaceutical Science
Carcinoma
100%
Estrogen Receptor
50%
Oncoprotein
50%
Single Chain Fragment Variable Antibody
50%
Cyclin D1
50%
Ribozyme
50%
Diseases
50%