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Function of normal and mutated γ-globin gene promoters in electroporated K562 erythroleukemia cells
M. J. Ulrich,
T. J. Ley
Division of Biology and Biomedical Sciences
Institute of Clinical and Translational Sciences
Siteman Cancer Center
Division of Oncology
Section of Stem Cell Biology
Research output
:
Contribution to journal
›
Article
›
peer-review
48
Scopus citations
Overview
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Medicine & Life Sciences
Leukemia, Erythroblastic, Acute
93%
Globins
89%
K562 Cells
84%
Genes
28%
Erythroid Cells
25%
Electroporation
24%
Transfection
17%
Nuclease Protection Assays
16%
Kanamycin Kinase
16%
vpr Genes
14%
Fetal Hemoglobin
13%
Erythroblasts
13%
Transcription Initiation Site
11%
DNA
11%
Plasmids
8%
RNA
7%
Cell Line
6%
Mutation
5%
Chemical Compounds
Electroporation
100%
Neomycin
49%
Globin
44%
Enhancer
42%
Hemoglobin
33%
Mutation
30%
Strength
22%
Environment
20%