Efficient hepatitis C virus particle formation requires diacylglycerol acyltransferase-1

  • Eva Herker
  • , Charles Harris
  • , Céline Hernandez
  • , Arnaud Carpentier
  • , Katrin Kaehlcke
  • , Arielle R. Rosenberg
  • , Robert V. Farese
  • , Melanie Ott

Research output: Contribution to journalArticlepeer-review

298 Scopus citations

Abstract

Hepatitis C virus (HCV) infection is closely tied to the lipid metabolism of liver cells. Here we identify the triglyceride-synthesizing enzyme diacylglycerol acyltransferase-1 (DGAT1) as a key host factor for HCV infection. DGAT1 interacts with the viral nucleocapsid core and is required for the trafficking of core to lipid droplets. Inhibition of DGAT1 activity or RNAi-mediated knockdown of DGAT1 severely impairs infectious virion production, implicating DGAT1 as a new target for antiviral therapy.

Original languageEnglish
Pages (from-to)1295-1298
Number of pages4
JournalNature medicine
Volume16
Issue number11
DOIs
StatePublished - Nov 2010

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