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Does Aging Activate T-cells to Reduce Bone Mass and Quality?
Rajeev Aurora,
Deborah Veis
Division of Bone & Mineral Diseases
Center of Regenerative Medicine
Roy and Diana Vagelos Division of Biology & Biomedical Sciences (DBBS)
Institute of Clinical and Translational Sciences (ICTS)
Rheumatic Diseases Research Resource-Based Center
Siteman Cancer Center
DBBS - Molecular Cell Biology
DBBS - Immunology
DBBS - Molecular Microbiology and Microbial Pathogenesis
Bursky Center for Human Immunology & Immunotherapy Programs (CHiiPs)
DBBS - Cancer Biology
Research output
:
Contribution to journal
›
Review article
›
peer-review
3
Scopus citations
Overview
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Keyphrases
T Cells
100%
Aging
100%
Bone Mass
100%
Bone Quality
100%
Bone Loss
66%
Age-dependent
50%
Osteoporosis
50%
Environmental Factors
33%
Genetic Factors
33%
Menopause
33%
Low-grade Inflammation
33%
Etiology
16%
Inflammation
16%
Infectious Diseases
16%
Cellular Senescence
16%
Long-lived
16%
Chronic Infection
16%
Microbiome
16%
Immune System
16%
Animal Model Study
16%
Aged Mice
16%
Human Studies
16%
Induced Effect
16%
Metabolic Disease
16%
Fracture Risk
16%
Osteoporosis Treatment
16%
Bone Changes
16%
Osteoporosis in Men
16%
Basal Rate
16%
Low Bone Mass
16%
Tissue-resident Memory T Cells
16%
Basal Bone
16%
Bone Microarchitecture
16%
Estrogen Loss
16%
Inflammaging
16%
Loss of Ovarian Function
16%
Senolytics
16%
Biochemistry, Genetics and Molecular Biology
T Cell
100%
Bone Mass
100%
Osteolysis
57%
Genetics
28%
Animal Model
14%
Human Study
14%
Metabolic Disorder
14%
Microbiome
14%
Immunity
14%
Memory T Cell
14%
Ovary Function
14%
Inflammaging
14%
Pharmacology, Toxicology and Pharmaceutical Science
Inflammation
100%
Infection
50%
Microbiome
50%
Chronic Inflammation
50%
Human Study
50%
Metabolic Disorder
50%
Immunology and Microbiology
T Cell
100%
Bone Mass
100%
Osteolysis
57%
Memory T Cell
14%
Ovary Function
14%
Immune System
14%
Microbiome
14%