Disruption of the Cdc42/Par6/aPKC or Dlg/Scrib/Lgl polarity complex promotes epithelial proliferation via overlapping mechanisms

Keyphrases

• Cdc42 100%
• Epithelial Ingrowth 100%
• Scrib 100%
• Polarity Complexes 100%
• Apical-basal Polarity 80%
• Epithelium 40%
• Polarity Proteins 40%
• Actomyosin Contractility 40%
• Tumor Necrosis Factor-α 20%
• Signaling Pathway 20%
• Tumor Suppressor 20%
• Downstream Signaling 20%
• In Cancer 20%
• Moesin 20%
• Early Events 20%
• Drosophila Melanogaster 20%
• Cancer Development 20%
• Disorganization 20%
• Defining Characteristic 20%
• Cortical Actin 20%
• Wing Disc 20%
• Actin Regulators 20%
• Oncogenic Viruses 20%
• Human Carcinoma 20%
• Junctional Complex 20%
• Non-autonomous 20%

Biochemistry, Genetics and Molecular Biology

• Muscle Contractility 100%
• Myosin 100%
• Tumor Necrosis Factor 50%
• Tumor Suppressor Protein 50%
• Moesin 50%
• Actin 50%
• Drosophila Melanogaster 50%
• Oncovirus 50%

Immunology and Microbiology

• Muscle Contractility 100%
• Myosin 100%
• Tumor Necrosis Factor 50%
• Oncovirus 50%
• Actin 50%
• Drosophila Melanogaster 50%