Different mechanisms regulate productive herpes simplex virus 1 (HSV-1) and HSV-2 infections in adult trigeminal neurons

Keyphrases

• Herpes Simplex Virus 2 (HSV-2) 100%
• HSV-2 Infection 100%
• Trigeminal Neurons 100%
• Productive Infection 75%
• Latency 25%
• Neuronal Subtypes 25%
• Transcription Start Site 25%
• Herpes Simplex Virus Type 1 (HSV-1) 25%
• Exon 1 25%
• Latency-associated Transcript 25%
• Trigeminal Ganglion 25%
• Cis-regulatory Elements 25%
• Regulatory Sequence 25%
• HSV Infection 25%

Neuroscience

• Herpes Simplex Virus 100%
• In Vitro 33%
• Exon 33%
• Transcription Initiation Site 33%
• Trigeminal Ganglion 33%
• Regulatory Sequence 33%

Biochemistry, Genetics and Molecular Biology

• Human Alphaherpesvirus 1 100%
• Regulatory Sequence 33%
• Regulatory Element 33%
• Exon 33%
• Transcription Initiation Site 33%