2 Scopus citations

Abstract

Glucocorticoid (GC) resistance is a key mechanism by which traditional asthma treatments become ineffective for patients, yet the molecular characteristics of the associated regulatory changes are largely unknown. Significant evidence suggests that crosstalk between p38 Mitogen Activated Protein Kinase (MAPK) and GC signalling pathways may contribute to this resistance. Based on a number of studies, a simplified GC signalling pathway model was developed and integrated with a pre-existing model of the p38 MAPK pathway. It is predicted that with experimental data, the validity and use of this model can be confirmed, corrections and updates can be made where necessary, and that through the two pathways' interface points the existence and scale of crosstalk can be examined.

Original languageEnglish
Title of host publication2012 IEEE Symposium on Computational Intelligence and Computational Biology, CIBCB 2012
Pages69-76
Number of pages8
DOIs
StatePublished - 2012
Event2012 IEEE Symposium on Computational Intelligence and Computational Biology, CIBCB 2012 - San Diego, CA, United States
Duration: May 9 2012May 12 2012

Publication series

Name2012 IEEE Symposium on Computational Intelligence and Computational Biology, CIBCB 2012

Conference

Conference2012 IEEE Symposium on Computational Intelligence and Computational Biology, CIBCB 2012
Country/TerritoryUnited States
CitySan Diego, CA
Period05/9/1205/12/12

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