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Decoupling dedifferentiation and G
2
/M arrest in kidney fibrosis
Benjamin D. Humphreys
Department of Medicine
Center of Regenerative Medicine
Roy and Diana Vagelos Division of Biology & Biomedical Sciences (DBBS)
Division of Nephrology
DBBS - Computational and Systems Biology
DBBS - Developmental, Regenerative and Stem Cell Biology
Institute of Clinical and Translational Sciences (ICTS)
Research output
:
Contribution to journal
›
Article
›
peer-review
3
Scopus citations
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2
/M arrest in kidney fibrosis'. Together they form a unique fingerprint.
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Keyphrases
Kidney Fibrosis
100%
Dedifferentiation
100%
G2-M Arrest
100%
Cyclin G1
100%
Fibrosis
50%
Fibrogenesis
50%
Kidney Disease Progression
50%
Cyclin-dependent Kinase 5 (Cdk5)
50%
Epithelial Dedifferentiation
50%
Knock-in Mouse Model
25%
Cell Cycle Arrest
25%
Therapeutic Target
25%
Kidney
25%
Therapeutic Approaches
25%
Cellular Mechanisms
25%
Renal Tubular Epithelial Cells
25%
Taguchi
25%
Atypical Cyclins
25%
Medicine and Dentistry
Cell Dedifferentiation
100%
Kidney Fibrosis
100%
Cyclin G1
100%
Fibrosis
50%
Disease Exacerbation
50%
Fibrogenesis
50%
Chronic Kidney Disease
50%
Cellular Mechanism
25%
Cell Cycle Arrest
25%
Cell Cycle Checkpoint
25%
Proliferating Cell Nuclear Antigen
25%
Biochemistry, Genetics and Molecular Biology
Proliferating Cell Nuclear Antigen
100%
Cell Dedifferentiation
100%
Fibrogenesis
40%
Knockout Mouse
20%
Cell Cycle Arrest
20%
Cell Cycle Checkpoint
20%