Characterization of arginylation branch of N-end rule pathway in G-protein-mediated proliferation and signaling of cardiomyocytes

Keyphrases

• Cardiomyocytes 100%
• G Protein 100%
• N-end Rule 100%
• ATE1 100%
• Arginylation 100%
• Vascular Defects 80%
• Cardiac Defects 60%
• Degrons 60%
• Myocardium 40%
• Overexpression 20%
• Signaling Pathway 20%
• Phospholipase C 20%
• Eukaryotes 20%
• Gene-deficient Mice 20%
• Cardiac Development 20%
• GTPase-activating Protein 20%
• Embryogenesis 20%
• Non-cell Autonomous 20%
• Proteolytic System 20%
• DNA Synthesis 20%
• Transferases 20%
• Proteolysis 20%
• RGS4 20%
• Transfer RNA (tRNA) 20%
• MEK1 20%
• Ventricular Septal Defect 20%
• Deregulation 20%
• Vascular Development 20%
• Aminoacyl-tRNA 20%
• Oxygen Sensitivity 20%
• N-terminal Amino Acids 20%
• Hypoxia-sensitive 20%
• Short-lived Protein 20%
• Protein Regulators 20%
• Mitotic Activity 20%
• Degron 20%
• Cardiac Signal 20%
• Non-cardiomyocytes 20%

Biochemistry, Genetics and Molecular Biology

• Cardiac Muscle Cell 100%
• N-End Rule 100%
• G Protein 100%
• N-Terminus 33%
• Eukaryote 16%
• Amino Acids 16%
• GTPase-activating Protein 16%
• Embryogenesis 16%
• Transferase 16%
• DNA Synthesis 16%
• RGS4 16%
• Aminoacyl-tRNA 16%
• Mitosis Rate 16%
• Arginine 16%
• Transfer RNA 16%
• Lecithinase 16%
• Proteolysis 16%