Abstract

In this issue of Immunity, Prinz and colleagues (2016) describe an unexpected mechanism underlying the role of type I interferon in the initiation of cognitive impairment and sickness behavior during viral infection through induction of chemokine CXCL10 in central nervous system epithelial and endothelial cells.

Original languageEnglish
Pages (from-to)718-720
Number of pages3
JournalImmunity
Volume44
Issue number4
DOIs
StatePublished - Apr 19 2016

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