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Autophagy and viral neurovirulence
Anthony Orvedahl
, Beth Levine
Institute of Clinical and Translational Sciences (ICTS)
Division of Infectious Disease
Bursky Center for Human Immunology & Immunotherapy Programs (CHiiPs)
Roy and Diana Vagelos Division of Biology & Biomedical Sciences (DBBS)
DBBS - Immunology
DBBS - Molecular Microbiology and Microbial Pathogenesis
Research output
:
Contribution to journal
›
Short survey
›
peer-review
75
Scopus citations
Overview
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Keyphrases
Viral
100%
Autophagy
100%
Neurovirulence
100%
Virus
22%
Herpes Simplex Virus Type 1 (HSV-1)
22%
Neurodegeneration
11%
Neuroprotection
11%
Intracellular Pathogen
11%
Viral Infection
11%
Antiviral Response
11%
Physiological Function
11%
Neuronal Cell Death
11%
Encephalitis
11%
Antiviral Role
11%
Herpesvirus
11%
Innate Immunity
11%
In Vivo Evidence
11%
Degradation Pathway
11%
Self-destruction
11%
Sindbis Virus
11%
Alphavirus
11%
Neuronal Dysfunction
11%
Cellular Adaptation
11%
Lysosomal Degradation
11%
Basal Autophagy
11%
Protein Turnover
11%
Cellular Survival
11%
Adaptation to Stress
11%
Viral Antagonism
11%
Viral Evasion
11%
Organelle Turnover
11%
Neuroscience
Autophagy
100%
Neurovirulence
100%
Herpes Simplex Virus
20%
Neurodegeneration
10%
In Vivo
10%
Cell Survival
10%
Neuronal Cell Death
10%
Encephalitis
10%
Neuroprotection
10%
Sindbis Virus
10%
Alphavirus
10%
Herpesviridae
10%
Medicine and Dentistry
Autophagy
100%
Human Alphaherpesvirus 1
20%
Antivirus Agent
20%
Virus
20%
Neurodegeneration
10%
Innate Immunity
10%
Cell Survival
10%
Cell Death
10%
Neuroprotection
10%
Pathogen
10%
Encephalitis
10%
Virus Infection
10%
Herpesviridae
10%
Alphavirus
10%
Sindbis Virus
10%
Protein Degradation
10%
Immunology and Microbiology
Autophagy
100%
Human Alphaherpesvirus 1
20%
Virus
20%
Cell Death
10%
Infectious Agent
10%
Viral Disease
10%
Cell Survival
10%
Herpesviridae
10%
Innate Immune System
10%
Sindbis Virus
10%
Alphavirus
10%
Encephalitis
10%
Protein Degradation
10%