Abstract
The evident defect in this disease is postsynaptic dysfunction due to antiacetylcholine receptor antibodies. The postsynaptic membrane is simplified, the number of acetylcholine receptors is reduced, and neuromuscular transmission is impaired. Antiacetylcholinesterase drugs still play a role in management. Newer options include prednisone, azathioprine, and cyclosporine.
| Original language | English |
|---|---|
| Pages (from-to) | 109-112+115+117+121-122+127-128+131 |
| Journal | Hospital Practice |
| Volume | 28 |
| Issue number | 1 |
| DOIs | |
| State | Published - 1993 |
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