Sepsis is a complex disease characterized by severe endothelial injury. In this issue of Cell Host & Microbe, Lubkin et al. (2019) reveal that an interaction between Staphylococcus aureus leukocidins and their cellular receptor DARC on endothelial cells leads to vascular injury, shedding light on pathogen-driven contributions to sepsis.

Original languageEnglish
Pages (from-to)351-353
Number of pages3
JournalCell Host and Microbe
Issue number3
StatePublished - Mar 13 2019


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