Abnormal presynaptic short-term plasticity and information processing in a mouse model of fragile X syndrome

Keyphrases

• Mouse Model 100%
• Information Processing 100%
• Plasticity Processing 100%
• Fragile X Syndrome 100%
• Short-term Plasticity 100%
• Fragile X Mental Retardation Protein 66%
• Knock-in Mouse Model 33%
• High-frequency Stimulation 33%
• Fmr1 Knockout 33%
• Pathophysiology 16%
• Axon 16%
• Spontaneous Excitatory Postsynaptic Current (sEPSC) 16%
• Cognitive Impairment 16%
• Intellectual Disability 16%
• Genetic Causes 16%
• Protein Synthesis 16%
• Autism 16%
• Dendrite 16%
• Novel Mechanism 16%
• Synaptic 16%
• Releasable 16%
• Calcium Influx 16%
• Presynaptic Mechanisms 16%
• Activity-dependent 16%
• Enhanced Response 16%
• Working Memory 16%
• Synaptic Vesicle Recycling 16%
• Long-term Depression 16%
• Long-term Plasticity 16%
• Hippocampal Synapses 16%
• Neural Function 16%
• Stimulus Train 16%
• Natural Stimuli 16%
• Vesicle Pools 16%
• Place Fields 16%

Neuroscience

• Information Processing 100%
• Fragile X Syndrome 100%
• Short-Term Potentiation 100%
• Fragile X Mental Retardation Protein 66%
• Cognitive Disorders 16%
• Protein Biosynthesis 16%
• Synapse 16%
• Dendrite 16%
• Calcium Transport 16%
• Presynaptic Mechanism 16%
• Synaptic Vesicle Recycling 16%
• Working Memory 16%
• Autism 16%
• Axon 16%
• Decision Making 16%
• Intellectual and Developmental Disabilities 16%