Abnormal glutamate homeostasis and impaired synaptic plasticity and learning in a mouse model of tuberous sclerosis complex

Keyphrases

• Tuberous Sclerosis Complex 100%
• Mouse Model 100%
• Synaptic Plasticity 100%
• Synaptic Learning 100%
• Glutamate Homeostasis 100%
• Hippocampus 66%
• Glutamate 66%
• Neuronal Death 66%
• Long-term Potentiation 66%
• TSC1 33%
• Functional Impairment 33%
• Seizure 33%
• Cell Death 33%
• Glia 33%
• Astrocytes 33%
• Hippocampal Slice 33%
• Excitotoxic 33%
• NMDA Antagonist 33%
• Extracellular Glutamate 33%
• Glutamate Transporter 1 (GLT-1) 33%
• Learning Impairment 33%
• Synaptic 33%
• Neocortex 33%
• Synaptic Function 33%
• Tetanic Stimulation 33%
• Associated Impairments 33%
• Microdialysis 33%
• Neurological Abnormalities 33%
• Glutamatergic 33%
• Behavior Learning 33%
• Learning Paradigm 33%
• Cell Death Assays 33%

Neuroscience

• Glutamic Acid 100%
• Synaptic Plasticity 100%
• Tuberous Sclerosis 100%
• Hippocampus 33%
• Cell Death 33%
• Neuronal Cell Death 33%
• Long-Term Potentiation 33%
• In Vivo 16%
• Learning Disabilities 16%
• Glia 16%
• Astrocyte 16%
• NMDA Receptor Antagonist 16%
• Glutamate Transporter 16%
• NADH Dehydrogenase (Ubiquinone) 16%
• Microdialysis 16%