Abstract
The posttranslational conversion of cysteine to Cα-formylglycine in the catalytic site of mammalian sulfatases is deficient in the rare but devastating disorder multiple sulfatase deficiency (MSD). Two papers in this issue of Cell (Dierks et al., 2003; Cosma et al., 2003) report the cloning of a gene responsible for this activity.
Original language | English |
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Pages (from-to) | 421-422 |
Number of pages | 2 |
Journal | Cell |
Volume | 113 |
Issue number | 4 |
DOIs | |
State | Published - May 16 2003 |