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β-Catenin induces T-cell transformation by promoting genomic instability
Marei Dose
, Akinola Olumide Emmanuel
, Julie Chaumeil
, Jiangwen Zhang
, Tianjiao Sun
, Kristine Germar
, Katayoun Aghajani
, Elizabeth M. Davis
, Shilpa Keerthivasan
,
Andrea L. Bredemeyer
, Barry P. Sleckman
, Steven T. Rosen
, Jane A. Skok
, Michelle M. Le Beau
, Katia Georgopoulos
, Fotini Gounari
Division of Cardiology
Center for Cardiovascular Research
Research output
:
Contribution to journal
›
Article
›
peer-review
57
Scopus citations
Overview
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Keyphrases
Catenin
100%
T Cells
100%
Cell Transformation
100%
Genomic Instability
100%
DNA Damage
50%
Thymocytes
50%
Recombination Activating Gene
50%
Lymphoma
33%
Binding Site
16%
Transcription Factor
16%
Apoptotic Genes
16%
Gene Expression Profile
16%
In Cancer
16%
Histone H3
16%
Mechanistic Insight
16%
Recombination Event
16%
T-cell Lymphoma
16%
Lysine
16%
Improved Survival
16%
Recurring
16%
RAG2
16%
Cell-type-specific Gene Expression
16%
DNA Double-strand Breaks
16%
Specific Transcription Factor
16%
Double-strand Break Repair
16%
Thymocyte Development
16%
Transformation Process
16%
Oncogenic Translocations
16%
Gene Recombination
16%
ChIP-seq Analysis
16%
H3K4me3
16%
Illegitimate Recombination
16%
Myelocytomatosis Oncogene
16%
Biochemistry, Genetics and Molecular Biology
Catenin
100%
T Cell
100%
Genome Instability
100%
Cell Transformation
100%
Thymocyte
66%
Recombination-Activating Gene
50%
DNA Damage
33%
Transcription Factors
33%
Fibrinogen
33%
Binding Site
16%
Gene Expression
16%
Carcinogenesis
16%
Histone H3
16%
Animal Model
16%
Transcription
16%
Genetic Recombination
16%
Myc
16%
Oncogene Myc
16%
RAG2
16%
Lysine
16%
Double-Strand DNA Break
16%
Immunology and Microbiology
T Cell
100%
Catenin
100%
Genomic Instability
100%
Cell Transformation
100%
Thymocyte
66%
Recombination-Activating Gene
50%
Transcription Factors
33%
DNA Damage
33%
Fibrinogen
33%
Gene Expression
16%
Binding Site
16%
Genetic Recombination
16%
Oncogene Myc
16%
Histone
16%
Lysine
16%
Double-Strand DNA Break
16%